Actinic Keratosis ICD-10: L57.0
Often searched as: rough scaly patch on face, crusty spot on forehead, scaly skin from sun damage, rough patch on scalp, dry rough spot on ear, skin that feels like sandpaper, pre-skin cancer treatment, precancerous skin spot…
Clinical urgency level
Systems Affected
Severity Levels
mild
One or few AKs on a low-risk site, thin and flat — responds well to cryotherapy or topical treatment (imiquimod, 5-FU).
moderate
Multiple AKs across large sun-damaged areas (field cancerization), some hypertrophic — requires field treatment (photodynamic therapy, topical 5-FU or imiquimod over a larger area).
severe
Hypertrophic or hyperkeratotic AK, or AK suspected of progressing to invasive SCC (thickening, ulceration, bleeding) — requires biopsy and possible excision.
Red Flags
- An AK that becomes thickened, raised, or nodular (possible SCC transformation)
- Bleeding or ulceration of a previously flat scaly patch
- Rapid increase in size over weeks
- Pain or tenderness developing in a previously asymptomatic lesion
- Multiple new AKs appearing rapidly in an immunocompromised patient
- Any AK on the lip (actinic cheilitis) — higher SCC transformation risk
Clinical Presentation
Actinic keratosis — rough, scaly erythematous patch on the forehead
Source: Wikimedia Commons (CC BY-SA 3.0 )
Actinic keratosis image gallery — DermNet NZ
View gallery on DermNet NZ (image gallery reference)When to See a Doctor
See a dermatologist if you have rough, scaly patches on sun-exposed skin — especially the face, scalp, ears, or backs of hands — that have been present for months without going away. AKs are treatable precancers; early treatment prevents SCC. Don't ignore a crusty, scaly spot just because it doesn't hurt.
Differential Diagnosis
- Squamous cell carcinoma in situ / Bowen's disease (well-defined, larger — biopsy needed)
- Seborrheic keratosis (waxy, stuck-on — benign)
- Psoriasis (well-defined silvery plaques)
- Discoid lupus erythematosus (scarring, depigmented center)
- Wart (verruca — viral, rougher texture)
- Superficial basal cell carcinoma (may mimic — biopsy key)
Comorbidities
- Squamous cell carcinoma (AK is the direct precursor lesion)
- Basal cell carcinoma (co-existing UV damage)
- Field cancerization (widespread UV-damaged skin with multiple AKs)
- Immunosuppression (organ transplant — AKs are numerous, aggressive, higher SCC risk)
- Xeroderma pigmentosum
- Albinism
Prognosis
Individual AKs have a low annual transformation rate to SCC (~0.1–1%), but the cumulative risk with many lesions over decades is meaningful. Approximately 60% of SCCs arise from AKs. Treated AKs have excellent outcomes — cryotherapy, photodynamic therapy, topical 5-FU, and imiquimod all achieve high clearance rates. However, new AKs continue to form on UV-damaged skin, making ongoing surveillance essential. Strict photoprotection is the most important long-term strategy.
Detailed Overview
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